Samheiti: Elektrólýtar í hægðum
Sýnataka, geymsla og sýnasending
Gerð sýnis : Fljótandi saur dreginn upp í þvagsýnarör mælingin er gerð í vökva.
Magn: 10 mL
Geymsla sýnis: Sent sem allra fyrst, annars kælir
Sýnasending: Hraðsending sem haldið er kaldri.
Heimildir
xbYbKFidNUsDbgWJiEZU_jpg.jpg Plaströr av polyprofylen (PP) 10 mL / Pp sterilt
| Remiss | Beställningsetikett | System | Faeces |
| Remissord | F-Kalium |
| Provtagning och hantering | Koniskt plaströr polypropen ("polypropylen"), 10 mL |
Flytande feces
Förvaras i kyla.
|
| Metod | Jonselektiv elektrod Indirekt mätning |
| Storhet | Substansmängdskoncentration | Enhet | mmol/L |
| NPU-Kod | NPU08629 | Ackrediterad | Nej |
| Remiss | Beställningsetikett | System | Faeces |
| Remissord | F-Natrium |
| Provtagning och hantering | Koniskt plaströr polypropen ("polypropylen"), 10 mL. |
Flytande feces.
Förvaras i kyla.
|
| Metod | Jonselektiv elektrod Indirekt mätning |
| Storhet | Substansmängdskoncentration | Enhet | mmol/L |
| NPU-Kod | NPU08649 | Ackrediterad | Nej |
Fróðleikur:
Stool osmotic gap is a calculation performed to distinguish among different causes of diarrhea.
It is calculated with the equation 290 − 2 * (stool Na + stool K).[1] The 290 is the value of the stool osmolality. The stool osmolality is usually not directly measured, and is often given a constant in the range of 290 to 300.[2]
A low stool osmolic gap can imply secretory diarrhea, while a high gap can imply osmotic diarrhea.[3] The reason for this is that secreted sodium and potassium ions make up a greater percentage of the stool osmolality in secretory diarrhea, whereas in osmotic diarrhea, molecules such as unabsorbed carbohydrates are more significant contributors to stool osmolality.
A normal gap is between 50 and 100 mosm/kg.[4]
High osmotic gap (>100 mosm/kg) causes of osmotic diarrhea include celiac sprue, chronic pancreatitis, lactase deficiency, lactulose, osmotic laxative use/abuse, and Whipple's disease.
Low osmotic gap (<50 mosm/kg) causes of secretory diarrhea include toxin-mediated causes (cholera, enterotoxigenic strains of E. coli) and secretagogues such as vasoactive intestinal peptide (from a VIPoma, for example). Uncommon causes include gastrinoma, medullary thyroid carcinoma (which produces excess calcitonin), factitious diarrhea from non-osmotic laxative abuse[5] and villous adenoma.